On day 3,7,10,21,30 after inoculation, cardiac systolic function, Vp ( peak velocity of aorta) and Vi ( flow velocity integral of aorta) were detected by echocardiography. 分别于接种后3d、7d、10d、21d、30d行超声心动图检查,测定主动脉血流峰值流速(Vp)、主动脉流速积分(Vi);
To use streptozotocin to produce the experimental diabetic rat model which has high expression of RAGE-mRNA in cardiac muscle and aorta. 建立链脲佐菌素糖尿病大鼠心肌及主动脉组织RAGE-mRNA高表达的实验动物模型。
Retinoic acid is a chemical teratogen which causes varieties of congenital cardiac malformations, such as overriding aorta, ventricular septal defect. 维生素A酸是常见的一类化学致畸因子,可引起各种心脏畸形,如主动脉骑跨、室间隔缺损等。
During a mean follow up of 32 months in 11 patients, cardiac function were improved in all patients after operation, one died of intracranial blooding, one suffered from recurrent dissection at the aortic arch and descending aorta. 11例平均随访32个月,心功能明显改善,晚期因颅内出血死亡1例,再发主动脉弓、降主动脉夹层动脉瘤1例。
Na~+/ Ca~ ( 2+) exchanger function changes in rats with cardiac dysfunction due to abdominal aorta stenosis 腹主动脉狭窄致心功能不全大鼠主动脉Na~+/Ca~(2+)交换功能变化
Results: Cardiac catheterization suggested severe pulmonary hypertension, anomaly of aortic arch, patent ductus arteriosus ( PDA) and communication between the adjacent parts of the ascending aorta and the pulmonary trunk in 4 patients. 结果:心导管检查4例均提示重度肺动脉高压、主动脉弓畸形、动脉导管未闭(PDA)及主&肺动脉根部有交通。
Methods: Pressure overload cardiac hypertrophy was induced by abdominal aorta constriction ( banding) in rats. 方法:结扎大鼠腹主动脉使之外径缩窄至0.6mm,导致心脏后负荷增高,形成心肌肥厚。
All fetus were showed severe cardiac hypertrophy, such as increased of cardiothoracic area ratio, dissymmetry of atrial and ventricle, ventricular wall hypertrophy, and decreased of the ventricular shortening fraction ( FS) and the total cardiac output of aorta and pulmonary artery ( CCO). 均示心脏显著肥厚扩大,表现为心胸比例增大,心房、心室的不对称及心室壁肥厚;心室缩短率(FS%)及心输出量(CCO)降低(P0.001)。
Conclusion The cardiac or aorta injuries need accurate early diagnosis. 结论心脏、大血管创伤应早期准确诊断;
The leftward deviation of the cardiac apex, aortic root dilation and abnormal ratio of pulmonary artery to aorta were helpful to the prenatal diagnosis of fetal tetralogy of Fallot. 主动脉根部内径增宽和肺动脉与主动脉内径比值减小亦有助于产前诊断。
Methods: The pressure overload cardiac hypertrophy model was produced by constriction of abdominal aorta. MAng ⅱ was measured by radioimmunoassay, and NOS by colorimetric method. 方法:本实验采用腹主动脉缩窄法建立大鼠压力负荷性心肌肥厚模型,用放免法测定MAng及比色法测定NOS含量。
Serum was taken for assays of cardiac troponion ⅰ( cTnI), CK and CK-MB at the time of preoperation, 8, 16, 24 and 48 hours after ascending aorta clamp removing. 另于术前、术后8h、16h、24h、48h取静脉血查心肌肌钙蛋白Ⅰ(cTnI)、CK及CK-MB。
Methods: ( 1) A rat model of cardiac hypertrophy in vivo was established by constriction of abdominal aorta. 方法:(1)用腹主动脉缩窄的方法建立大鼠心肌肥厚的体内模型。
In 8 anaesthetized male adult dogs the left cardiac ventricular pressure ( LVP), the rate of change of pressure with respect to time ( dp/ dt), the blood flow in the ascending aorta and in the left circumflex coronary artery were recorded. 成年雄狗8条,在麻醉开胸条件下,记录左室压(LVP)、室内压变化率(dp/dt)、升主动脉内压力和血流、左冠脉旋支血流。
Methods The range of C wave of cardiac impedance differential plethysmography by six pairs of electrodes at the same time was observing, with dog's aorta and lung artery broken and narcotized. 方法用阻断麻醉狗主动脉和肺动脉的方法,同步观察六对方片电极上心阻抗微分图C波幅度的改变。
Methods Between Jan 2000 to Sep 2004, 310 patients were operated with cardiac valve replacement, including mitral valve in 148, aorta valve in 36, and combined mitral valve and aorta valve in 126. Results Fourteen patients were died in the early of operation. 方法2000年1月~2004年9月行瓣膜置换术310例,其中二尖瓣置换术148例,主动脉瓣置换术36例,二尖瓣与主动脉瓣联合置换126例。
The cardiac output was measured with the magnetic volumetric meter at the root of aorta. 用电磁流量计探头放置到主动脉根部,测量心输出量;
Constrction of abdominal aorta can induce cardiac hypertrophy in rats, SIM can inhibit abdominal aorta banding induced cardiac hypertrophy, the mechanism is related to the inhibition of JAK/ STAT signal pathway. 腹主动脉缩窄可致大鼠心肌肥厚。SIM对心肌肥厚具有明显的防治作用,其作用机制与抑制JAK/STAT信号通路激活有关。